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The dream of Scottish independence died at 6:17 a.m. (GMT), when an exhausted Alex Salmond, leader of the Scottish National Party (SNP) and the moving force behind independence, conceded defeat.
"I accept the verdict of the people," he told a crowd of tearful supporters. "And I call on all the people of Scotland to follow suit."
The final tally, delivered by chief recording officer Mary Pitcaithly, was 55.4 percent for No and 44.6 for Yes.
It was the end of a boisterous and sometimes rancorous two-year campaign that engaged Scotland and the rest of the British Isles like no other political issue since the end of World War II.
"It's fantastic," said Hamish Macarthur, a retired chemical engineer, who spent the past month campaigning in his native Stirling. "But I'm not surprised. The Scottish people are not stupid, and what the nationalists offered was simply not credible."
"I Feel Very Emotional"
For the Yes camp, it meant heartbreak. "It's sad for the brave nation of Scotland to succumb to fear," sighed Audrey Gilles, a former care worker from Hawick, in the Border country. "I feel very emotional."
The first count came in Clackmannanshire, in central Scotland, with a 7 percent win for the Better Together campaign. From then on, defeat rained down like hammer blows on the head of Salmond, stuck at home in Aberdeen in heavy fog.
Inverclyde, an industrial area west of Glasgow, which the Yes campaign needed to win, went to No. So did East Renfrewshire, Midlothian, Perth and Kinross, and many others.
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In Record Turnout Demographics Shape Scotland's Emphatic No Vote
AFP Ocean-threatened Marshall's leader posts climate video plea
Majuro (Marshall Islands) (AFP) - Marshall Islands President Christopher Loeak was forced to heighten the seawall protecting his home last year, but says the year-old defences are now barely enough to protect his family from a "climate emergency".
PRE Friday, ahead of UN Secretary General Ban Ki-moon's climate summit in New York City next week.
Standing outside his home in the capital Majuro next to the heightened seawall, Loeak tells the camera that it is "barely enough to protect my family from the encroaching waves."
"For the Marshall Islands and our friends in the Pacific, this is already a full-blown climate emergency," Loeak said.
He will join more then 100 heads of state at the September 23 forum, which he hopes will galvanise support to build "the greatest climate change alliance" the world has seen.
The United Nations is seeking to limit global warming to two degrees Celsius (3.6 degrees Fahrenheit) over pre-industrial levels, but scientists say current emission trends could hike temperatures to more than twice that level by century's end.
While US President Barack Obama is to outline his vision for reining in greenhouse gas emissions, Chinese President Xi Jinping and Indian Prime Minister Narendra Modi are among a number of prominent world leaders who will be no-shows at next week's meeting.
UN climate envoy Mary Robinson believes the summit will see the world begin to seriously tackle global warming ahead of a crucial conference in Paris next year.
"The message from the climate summit and the message going forward to Paris is that it's not business as usual with a little bit of green attached," Robinson said recently.
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LIFEBOATS were on standby in the Western Isles tonight after thick fog threatened to ground planes taking ballot boxes to the count.
It was feared the plane being used to ferry ballot boxes around the islands might not be able to land at Stornoway - where all the isles' votes will be gathered.
It is the only plane that is being used to carry votes in the whole of Scotland.
Meanwhile, many postal votes were stuck on the mainland this morning as the mail plane - which was due in at Stornoway at 7.20am - could not land because of the fog, before eventually it did at 2pm.
The Western Isles is in the running to be the first of 32 regions in Scotland to declare the local result, looking at a 1.30am announcement
But the blanket of fog lying over the islands earlier today threatened plans to use a chartered plane to fly ballot boxes, containing around 4000 votes from the North and South Uist and Barra to the count centre in Stornoway.
Calmac Ferries and RNLI lifeboats around the islands were put on stand-by if fog left the planes unable to take off and land.
But this afternoon Keith Campbell, managing director of plane company Jet Logic, said he hoped flights would go ahead as planned.
The Britten Norman Islander will come from Oban and land at Benbecula before 9pm and is due to take off with the ballot boxes and an accompanying official at just before midnight. It should arrive in Stornoway about 45 minutes later.
"We are confident we can get the plane into Benbecula - the problem may be Stornoway. But the latest weather forecast suggests that it should be OK," said Mr Campbell from the Edinburgh-based firm.
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Independence referendum: Lifeboats on standby in Western Isles after fog threatens to delay count
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Newswise The Genetics Society of America today announced new appointments to the editorial board of its flagship journal GENETICS. The recent additions complement the renewed focus of the peer-reviewed, peer-edited journal in the areas of genomics, human genetics, and methods, among other fields.
The newest members of the board bring cutting-edge expertise and reflect the changing, interdisciplinary landscape of our field, while exemplifying GENETICS reputation for rigorous scholarship, said Mark Johnston, Editor-in-Chief of GENETICS and Professor and Chair, Department of Biochemistry and Molecular Genetics, University of Colorado School of Medicine. Theyre already adding to the momentum of the journals transformation over recent years.
Two former Associate Editors have also taken on Senior Editor roles to lead the journals expanded coverage of methods and technology development:
New methods and technologies often drive important discoveries in genetics, and the journal welcomes papers that describe these new approaches, said Fields.
The new Statistical Genetics and Genomics section will handle articles describing statistical methods, which were formerly published in the Methods, Technology, and Resources section.
Statistics is an integral part of our field, so its only fitting that it has a dedicated section and its own team of Associate Editors, said Churchill.
The new Senior Editors are joined by several Associate Editor appointments this year:
New Editor Details: Hugo J. Bellen Baylor College of Medicine & Howard Hughes Medical Institute GENETICS Associate Editor, Developmental and Behavioral Genetics http://flypush.imgen.bcm.tmc.edu/lab/index.html
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New Editors Join GENETICS, the Flagship Journal of the Genetics Society of America
Heidi Richmond walks her dog, Grizz, who is being treated with the canine melanoma vaccine. The medication is a form of immunotherapy, teaching the dog's immune system to fight the cancer.
Ray Boone, Deseret News
MURRAY The lifelong bonding between humans and dogs is eloquent. Dogs are loved as members of families. And just like family members, when they become ill, owners want them to have the best medical care.
It appears dogs and humans are much more alike genetically than was originally believed, and what's saving their lives could save human lives as well.
In fact, researchers are "going to the dogs," so to speak, to form a unique partnership.
At Cottonwood Animal Hospital in Murray, Heidi Richmond's dog "Grizz" is being treated with a vaccine that's a form of immunotherapy. The treatment is approved only for oral melanomas in dogs, but designed from human genetics. Veterinarian Nathan Cox said this kind of match-up intrigues researchers.
"The genetics of cancer in dogs is very similar to what it is in people," Cox said, "and that allows us a baseline to be able to study cancer in an alternate species."
With traditional therapy, a dog with melanoma undergoes surgery or radiation to debulk the tumor, he said. Dogs' average lifespan after treatment, without the vaccine, is usually less than six months.
"It's (the vaccine) really changed the game," Cox said. "It's been more effective than chemotherapy has been for oral melanoma in dogs."
For Grizz and other dogs with cancer, this human genetic product is different enough to trigger an immune response, but similar enough to the dogs' own melanoma to cross react, training the immune system to attack the cancer cells.
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Mutations in the human retinoblastoma protein gene are a leading cause of eye cancer. Now, Michigan State University scientists have turned to fruit fly eyes to unlock the secrets of this important cancer gene.
In a paper featured on the cover of the current issue of the Journal of Biological Chemistry, Michigan State University researchers provide the first detailed examination of a set of mutations similar to those present in the human cancer gene, said Irina Pushel, MSU undergraduate and co-author.
"By systematically evaluating mutations of increasing severity, we now have a model to better predict how we think the protein will react with each mutation," said Pushel, who co-authored the paper with Liang Zhang, lead author and MSU graduate student, and Bill Henry and David Arnosti, MSU molecular biologists. "We're trying to understand the protein, not even in the specific context of cancer, but rather studying how it interacts within the cell, how it interacts with DNA."
The protein, retinoblastoma, would appear to play a key role in everything. When it's healthy, it helps control cell growth and development. If absent, the organism would die. In its abnormal state cells can overgrow, as seen in cancer, or undergo premature death, as in other human diseases.
Since fruit flies are essentially tiny people with wings, in terms of genetics, these model organisms can play a key role in advancing human medicine.
"If we find one of these mutations in a human, then we can predict what will happen with the protein, such as folding incorrectly," Pushel said. "This isn't going to immediately lead to a new drug to treat cancer. However, we have to know how the protein works before we can develop a drug to fix it. Future medicines will be built upon models such as this, though that is years away."
Previous work has shown that a specific part of this protein plays a role in regulating other genes. In this study, the team modified some of the known important parts of this region of retinoblastoma.
Boosting levels of even standard, or wild-type, protein altered fruit flies eyes and wings. However, when levels of the mutated protein began to climb, deformations were consistent and dramatic.
While a cancer treatment based on this finding may be years away, the insight and understanding into cell development and gene regulation is immediate, Pushel said.
"That's the cool thing about basic research; it may not lead directly to the creation of a new drug, but it helps decipher the genetic code, which for each person controls the unique pattern of how they grow and how they develop -- that's amazing," she said. "It will have many impacts, from understanding development to personalized medicine."
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PUBLIC RELEASE DATE:
18-Sep-2014
Contact: Wolfgang Enard enard@bio.lmu.de 49-089-218-074-339 Ludwig-Maximilians-Universitt Mnchen
The ability to acquire and creatively manipulate spoken language is unique to humans. "The genetic changes that occurred over the past 6 million years of human evolution to make this possible are largely unknown, but Foxp2 is the best candidate gene we now have," says Wolfgang Enard, Professor of Anthropology and Human Biology at LMU. In his efforts to understand the molecular biological basis of language Enard has now taken an important step forward. The results of his latest study, undertaken in collaboration with scientists at several universities, including the Massachusetts Institute of Technology in Cambridge and the Max Planck Institute for Evolutionary Anthropology, have recently appeared in the journal Proceedings of the National Academy of Sciences (PNAS).
The human homolog of Foxp2 codes for a protein a so-called transcription factor that regulates the activity of hundreds of genes expressed in various mammalian cell types. Individuals who carry only one functional copy of the gene instead of the usual two experience specific difficulties in learning to speak and in language comprehension. "Genetic mutations that occurred during the 6 million years since our lineage diverged from that of chimpanzees have resulted in localized alterations in two regions of the Foxp2 protein. That is quite striking when one considers that the normal mouse version differs from that found in chimps by only a single mutation, although these two species are separated by over 100 million years of evolution. The question is how the human variant of this transcription factor contributes to the process of language acquisition," says Enard.
Enard and his coworkers had previously shown that the alterations in the human gene for Foxp2 specifically affect certain regions of the brain. When the two human-specific substitutions were introduced into the mouse version of the gene, he and his team observed anatomical changes exclusively in two neuronal circuits in the basal ganglia of the mouse cortex, which are involved in the control of motor function. "These circuits play a crucial role in the acquisition of habitual behaviors and other cognitive and motor capabilities," Enard explains.
Conscious and unconscious learning processes
In their latest work with the same mouse model, Enard and his collaborators found that, under certain conditions, the human version of Foxp2 actually enhances learning. "We have shown for the first time that the evolved alterations in the human gene have an effect on learning ability. The human version modifies the balance between declarative and motor neuron circuits in the brain. As a result, the mice take less time to associate a given stimulus with the appropriate response, and hence learn more rapidly," says Enard.
Learning to speak clearly requires interactions between conscious "declarative" knowledge and the unconscious effects of repetitive stimulation of particular patterns of neural activity. "As we learn, the underlying neuronal processes become automated, they are converted into routine procedures, enabling us to learn faster," Enard explains. Using various tests, the researchers demonstrated that the human-specific mutations enhance cooperative interactions between the two affected circuits in the basal ganglia of the mouse brain. "The human variant of the Foxp2 gene modulates the associative and sensorimotor nerve connections formed, as well as levels of the neurotransmitter dopamine in the basal ganglia, during the learning process. The increased ability to switch between conscious and unconscious forms of learning may play a role in the acquisition of language," Enard concludes.
Foxp2 is the only gene so far that has been shown to be directly associated with the evolution of language, and studies of Foxp2 function promise to throw new light on the evolution of the human brain. The mutation that first revealed the link with language was discovered in a kindred, many of whose members displayed severe speech difficulties, primarily as a consequence of defective control of the muscles of the larynx, the lips and the face.
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PUBLIC RELEASE DATE:
18-Sep-2014
Contact: Raeka Aiyar press@genetics-gsa.org 202-412-1120 Genetics Society of America @GeneticsGSA
Life is rarely simple. From crop yields to disease risks, the biological characteristics people care most about are usually those considered "complex traits." Just as for heightthe textbook example of a complex traitattributes like risk for a particular human disease are shaped by multiple genetic and environmental influences, making it challenging to find the genes involved. To track down such genes, geneticists typically mate two individuals that differ in key waysfor example, a large mouse and a small mouseand then study their descendents, looking for genes that tend to be inherited with the trait value of interest. But this method only implicates a broad genomic region, and the identities of the crucial gene/s often remain a mystery.
Now, geneticists are embracing a powerful approach that pinpoints more precise areas of the genome by founding the breeding population with multiple, genetically diverse parents. To encourage innovations in this rapidly developing field, the Genetics Society of America journals GENETICS and G3: Genes|Genomes|Genetics today published the first articles in an ongoing special collection on mapping complex trait genes in multiparental populations.
The 18 articles describe methods and applications in a wide range of organisms, including mice, fruit flies, and maize. Among the advances reported are the creation of a multiparental population of wheat, methods for use with the Diversity Outbred and Collaborative Cross mouse populations, and the identification of nicotine resistance genes in fruit flies. The power of the approach for disease genetics is highlighted in an article describing how a multiparental rat population was used to find a human gene variant that affects insulin levels.
"These collections of multiparental strains are extremely powerful and greatly accelerate discovery. For example, in one of the articles, researchers report using a multiparental population to rapidly identify fruit fly genome regions associated with the toxicity of chemotherapy drugs. The authors could then examine these regions to find several candidate causative genes," said Dirk-Jan de Koning, Professor at the Swedish University of Agricultural Sciences, Deputy Editor-in-Chief, Complex Traits, at G3, and an editor of the new collection. "Using standard two-parent crosses, they would have been stuck with unmanageably large regions each containing hundreds or even thousands of candidate genes."
Because the field is so new, geneticists are still developing the best methods for creating and analyzing multiparental populations. "This collection will move the field forward by stimulating discussion between different disciplines and research communities," said Lauren McIntyre, Professor at the University of Florida, and an editor of the collection. "To help foster this ongoing exchange, the collection will continue to publish new articles, and all associated data will be freely available."
In an editorial, McIntyre and de Koning describe how the idea for the multiparental populations collection was born and how scientific society journals like GENETICS and G3 can advance new research fields.
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Want to link genes to complex traits? Start with more diversity
WEDNESDAY, Sept. 17, 2014 (HealthDay News) -- Present-day Europeans are the descendants of at least three groups of ancient humans, according to a new study.
Previous research suggested that Europeans descended from indigenous hunter-gatherers and early European farmers. But, a new genetic analysis involving ancient bone samples revealed they are also the descendants of Ancient North Eurasians. Nearly all present-day Europeans have genetic material from this third ancestral group, researchers from Harvard Medical School said.
In conducting its investigation into Europeans' heritage, the team of researchers collected and sequenced the DNA of more than 2,300 people currently living around the world. They also examined DNA from nine ancient humans from Germany, Luxembourg and Sweden.
The ancient samples were taken from the bones of eight hunter-gatherers who lived about 8,000 years ago, and one farmer who lived about 7,000 years ago.
"Ancient DNA has emerged as a powerful technology that makes it possible to go back in time to understand how people in the past relate to people today," study co-senior author, David Reich, professor of genetics at Harvard Medical School, said in a university news release.
About 7,500 years ago in Europe, agriculture from the Near East brought early farmers into contact with hunter-gatherers who had been living in Europe for tens of thousands of years. Nearly all Europeans are the result of the mixing of these two ancient populations.
"There was a sharp genetic transition between the hunter-gatherers and the farmers, reflecting a major movement of new people into Europe from the Near East," noted Reich.
The study's authors found, however, Ancient North Eurasians also contributed DNA to present-day Europeans. Ancient North Eurasians also likely contributed DNA to people who crossed the Bering Strait into the Americas more than 15,000 years ago, according to the researchers.
"Nearly all Europeans have ancestry from all three ancestral groups," explained the study's first author, Iosif Lazaridis, a research fellow in genetics in Reich's lab.
"Differences between them are due to the relative proportions of ancestry. Northern Europeans have more hunter-gatherer ancestry -- up to about 50 percent in Lithuanians -- and Southern Europeans have more farmer ancestry," Lazaridis said in the news release.
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Europeans Are Descendants of at Least 3 Ancient Human Groups: Study
Dogs, humans attack cancer together
By Ed Yeates
September 18th, 2014 @ 7:02pm
SALT LAKE CITY It appears dogs and humans are much more alike genetically than we believed, and what's saving their lives could save our lives as well. In fact, researchers are "going to the dogs," so to speak, to form a unique partnership.
And why shouldn't they?
The lifelong bonding between humans and dogs is eloquent. We love them as members of our families. Their loyalty to us is boundless. And now, that bond goes much deeper.
At Cottonwood Animal Hospital, Heidi Richmond's dog Grizz is being treated with a vaccine that's a form of immunotherapy. The treatment is approved only for oral melanomas in dogs, but designed from human genetics. Veterinarian Nathan Cox says this kind of match-up intrigues researchers.
"The genetics of cancer in dogs is very similar to what it is in people, " he said. "That allows us a baseline to be able to study cancer in an alternate species."
For Grizz and other dogs with cancer this human genetic product is different enough to trigger an immune response but similar enough to the dogs own melanoma to cross react training the immune system to attack the cancer cells.
"We have dogs living out past three to four years with the vaccine, so it's more than doubled survival times and in some dogs," Cox said. "It's actually resulted in a cure for their disease."
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Newswise The Genetics Society of America today announced new additions to the editorial board of its peer-reviewed, peer-edited journal G3: Genes|Genomes|Genetics. Since the journals launch in June 2011, its editorial board of academic experts has been instrumental in shaping G3 into an important forum for the publication of useful genetics findings and resources.
As the breadth of research published in the journal continues to grow, Editor-in-Chief Brenda Andrews, PhD, has appointed two new Deputy Editors-in-Chief, who will contribute to the oversight of key sections:
Also, Stephen Wright, PhD, University of Toronto, has been appointed as a new Senior Editor for Population and Evolutionary Genetics and Genomics and will spearhead the journals efforts to strengthen coverage of in this area.
The new Deputy Editors-in-Chief and Senior Editor are joined by several new Associate Editor appointments to the editorial board this year:
Genetics is a fast-paced field. The expanded editorial board will help us keep up with the growing volume and diversity of research being submitted to G3, said Brenda Andrews, G3: Genes|Genomes|Genetics Editor-in-Chief and Professor and Chair of the Banting & Best Department of Medical Research, University of Toronto.
G3 was created by the Genetics Society of America to meet the critical and growing need of the genetics community for rapid review and publication. The journal offers an opportunity to publish the puzzling finding, useful dataset, or highly focused research that may not have been submitted for publication due to a lack of perceived impact.
New Editor Details:
Eduard Akhunov Kansas State University G3 Associate Editor
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Both Hands on the Wheel - Ministry Health Care Kohl #39;s Cares PSA
A joint effort of Ministry St. Joseph #39;s Children #39;s Hospital and Kohl #39;s Cares, this TV spot dramatically warns teen drivers about the dangers of distracted driving, specifically texting and...
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Both Hands on the Wheel - Ministry Health Care & Kohl's Cares PSA - Video
Forum Club Rep. Deutch on Health Care
With all eyes waiting to see if the U.S. Supreme Court strikes down all or part of President Obama #39;s Affordable Care Act, the Forum Club brought together two area congressmen one Republican,...
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Forum Club Rep. West on Health Care
With all eyes waiting to see if the U.S. Supreme Court strikes down all or part of President Obama #39;s Affordable Care Act, the Forum Club brought together two area congressmen one Republican,...
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WEBINAR: Moving The Industry/Health Care Professional Partnership Forward
September 16th, 12:30 14:00 CET (11:30 13:00 UK) In preparation for the implementation of The EFPIA Code outlining the disclosure of transfers of value to healthcare professionals,...
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WEBINAR: Moving The Industry/Health Care Professional Partnership Forward - Video
The health care industry has long been a laggard in adopting technology, but that will soon change as the challenge of aligning doctors, insurance companies and patients is figured out.
Health care has always been 30 years behind adopting technology compared to other industries, said Chris Gordon, managing director at investment firm Bain Capital, during the discussion Wednesday at The Economists Health Care Forum in Boston.
U.S. government efforts to spur the use of electronic health records created an infrastructure for storing patient data. The next challenge, Gordon said, is delivering meaningful results to people, an issue that Apple, Google and other major technology firms can potentially solve as they enter the health care space.
Now that that information is available and can flow, youll see big players come on and well be able to bridge that gap, he said. The building blocks are in place to see that over the next decade.
Disruption is finally coming to health care, said Unity Stokes, president and founder of StartUp Health, a startup accelerator aimed at helping health care startups grow, who noted that entrepreneurs from outside health care are entering the market.
But this upheaval wont follow the traditional path taken by startups looking to shake up a market. Instead of going around incumbent players, health care startups will work with stakeholders like the government and care providers.
In health care, you need to work with stakeholders to navigate the system, Stokes said.
Combining smartphone technology, human genome sequencing and the Internet can have a quantum-leap effect on health care, but only if these disruptive technologies are integrated into existing workflows, said Anita Goel, chairman and CEO of Nanobiosym.
Her company develops portable testing systems to quickly identify diseases such as HIV. Nanobiosym customizes mobile apps to fit with current systems so the technology can get outside the lab and have an impact.
To assuage data security concerns, people need to see how sharing health information can have positive effects on their health, Stokes said. Allowing a doctor access to health information, for example, can lead to the early detection of possible health problems, he said.
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As health care digitizes, consumer technologies will have greatest impact, says panel
Amarillo, TX - A collaboration of area doctors, physicians and nurse practitioners is cutting local Medicare costs by millions.
About two years ago, the Amarillo Legacy Medical Accountable Care Organization (ALMA) formed here in Amarillo to streamline medicare services. Thursday they received results of how their efforts are keeping more patients out of the hospital and in turn, saving taxpayer's money.
ALMA serves 10,876 Medicare patients across the Panhandle.The organization created strategies for 109 local health care providers to work together to prevent illnesses before they cause costly hospitalizations.They did this by improving access to care, coordinating health care between providers and by exchanging health care information.
"This was kind of an experiment of sorts that for the first time competing groups could work together and design programs that would benefit their mutual patients in order to improve quality and save money," explained ALMA CEO and Medical Director Dr. William Biggs.
So far, Dr. Biggs said the collaboration experiment is working. Hospital admissions for Medicare patients in Amarillo are down 23 percent since 2012.
Overall, the actual cost per patient in 2013 compared to the expected cost saved Medicare $461 per patient. That may not seem like a lot, but when you multiply that number by the total amount of Medicare patients they served last year, it adds up to almost $5 million in savings. "We thought we might be able to save about $1 million for the year, so $5 million in our mind is a huge amount of money. It blows away our expectations," said Dr. Biggs.
Medicare does return a portion of the savings to ALMA's group of providers as an incentive to participate. They will use part of that money to improve their efficiency and save even more federal money. "We plan basically to use that to reinvest and expand the program," said Dr. Biggs. "We're going to expand our care management to have more staff that will do that, we're going to extend our health information exchange to include the hospitals and additional medical groups, and we hope to have other medical groups join us in the coming year so that we'll have more doctors included."
So far, Accountable Care Organizations across the nation said they have saved more than $372 million in Medicare services.
To view the full presentation on Amarillo's Accountable Care Organization, click here.
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Ramsay's Christopher Rex. Photo: Rob Homer
Overall director activity declined substantially this week thanks to reduced director selling.
The scorecard registered $6 million to $12 million in favour of sellers, compared with buying of $4 million and sales of $65 million previously.
Ramsay Health Care chief Christopher Rex headed the sellers' list when he peeled off 6 per cent of his stake, collecting $3.7 million.
He also picked up about $543,000 of stock under an incentive plan and he is now sitting on a handy $55 million of stock.
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The estate of the late Paul Ramsay this month raised about $215 million through the sale of shares at $49 a piece.
Elsewhere, Donald McLay, chairman of Credit Corp, continued selling shares.
He has sold about $5 million worth of shares in the debt collection group in the last couple of months.
At last count, his interests held more than $16 million of stock.
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