Paul Ryans worst nightmare: Heres the real way to cut poverty in America

Poverty is back in the news, for several reasons. The first is the 50th anniversary of President Lyndon Johnsons 1964 War on Poverty speech. In addition, Republican congressman and 2012 vice-presidential candidate Paul Ryan has released a much-criticized report about federal poverty programs. In 2012 the Romney-Ryan ticket suffered from Mitt Romneys dismissive comments about the 47 percent and conservative caricatures of the poor as welfare-dependent moochers and takers. Ryans attempt at a version of what George W. Bush called compassionate conservatism appears to be an effort at rebranding the right as something other than an alliance of Have-Lots and Have-Somes against Have-Nots.

Public debate about poverty typically focuses on the causes of poverty, rather than the cures. The causes of poverty are many and various. You may be poor because you are the child of poor parents; or because you grew up in an economically distressed urban or rural region; or because you were bankrupted by unexpected medical bills; or because you lost all your money gambling on imaginary real estate in Second Life (this actually occurred, in a case of which I know). Because poverty has multiple causes, policies must be equally numerous, if the goal is to avert or prevent poverty in the future.

But its not necessary to avert or prevent poverty in the future in order to cure the poverty that already exists in the present, for whatever reason. Let me illustrate this point with an example. The treatment of victims of gunshot wounds in the emergency room may be identical even though one gunshot wound was caused by a shooting in the course of a robbery, another by a failed suicide attempt and a third by reckless play with a firearm. Doctors and nurses can treat the victims of the gunshot wounds now, while leaving others to propose better policing, better suicide-prevention counseling and better firearm safety training in the future.

Fortunately, drastically reducing existing poverty in the U.S. is not a difficult intellectual problem, even though it is a difficult political problem. With sufficient political will, we could slash existing poverty in the U.S. very quickly, while simultaneously trying to prevent as much poverty as possible in the future. Some public policy problems, like averting global warming or regulating shadow banking, are incredibly complex. By comparison, antipoverty policy is simple.

We know exactly what we need to do to radically reduce poverty in America. We know that it could be done, and we know how to do it, because many other First World democracies have slashed poverty already.

Among developed nations, the U.S. is an outlier in having a high proportion of its population living in poverty. Among the 34 member nations of the Organization for Economic Cooperation and Development (OECD), in 2010 on average 11.1 percent of the population suffer from relative income poverty. In the U.S. , however, the number is 17.4 percent. Among developed countries, only Chile (18%), Turkey (19.3%), Mexico (20.4%) and Israel (20.9%) have more of their people living in poverty, according to the OECD.

The low-poverty nations tend to be Scandinavian countries like Sweden (9.1%), Norway (7.5%), Finland (7.3%) and Denmark (6.0%). Some on the right argue that it is wrong to compare small, relatively homogeneous countries with a giant, pluralistic, continental society like the U.S. Others argue that the English-speaking countries as a whole are willing to tolerate more poverty and inequality than the Nordic social democracies.

The numbers dont support these arguments. Among the most populous Western states are France (7.9%) and Germany (8.8%), both of which have around half as many people in poverty as the U.S., notwithstanding their own growing immigrant populations. And while all English-speaking countries tend to be less statist than continental European societies, all of the other anglophone nations have considerably less poverty than the U.S., including Australia (14.4%) and Canada (11.9 %). Indeed, three English-speaking countries Ireland (9.0%), the UK (10.0 %) and New Zealand (10.3%) have fewer citizens in poverty than the OECD average in 2010 of 11.1%.

How do other countries do it? They dont necessarily have fewer poor people to begin with. According to an OECD study, with respect to pre-tax, pre-transfer poverty, the U.S., at 13, ranked in the middle of 26 high-income nations. When it comes to post-tax, post-transfer poverty, however, the U.S. was nearly the worst, second only to Israel.

The difference is entirely the result of government social spending on the poor mostly in the form of transfer payments, like public pensions, unemployment insurance, child subsidies and/or wage subsidies. Many other developed democracies start out with lots of poor people, just like the U.S. But the countries with big welfare states remove most of them from poverty. The American welfare state does lower the poverty rate but not enough. The American welfare state is way too small to be effective in doing its job of lowering poverty.

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Paul Ryans worst nightmare: Heres the real way to cut poverty in America

An Ontario Libertarian Minute #5: "Give the jobs back to the job makers." – Video


An Ontario Libertarian Minute #5: "Give the jobs back to the job makers."
Ontario has lost a great many manufacturing jobs over the past ten years. How would Ontario Libertarians create jobs? Part 3 Created and edited by Scott Mars...

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An Ontario Libertarian Minute #5: "Give the jobs back to the job makers." - Video

New Scrouge – Magnificent "2014 Virgin Islands Soca" (Prouced By Soto Productions) – Video


New Scrouge - Magnificent "2014 Virgin Islands Soca" (Prouced By Soto Productions)
Subscribe to Channel Here: http://tiny.cc/julianspromos | Produced by Soto Productions. Mixed and Mastered by: 3p Productions Twitter: @PamPam_3p | Twitter :...

By: JulianspromosTV | Soca Music

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New Scrouge - Magnificent "2014 Virgin Islands Soca" (Prouced By Soto Productions) - Video

Tiwi Islands: where AFL is a way of life

A woman in her seventies, bent with age, is swathed head to toe in blue and she's hollering so loud I temporarily lose hearing in my left hear.

"Bring it home, boys, BRRRRRING IT HOOOOOOOME!"

She's barracking for the Tuyu Buffaloes, and her screams are rewarded minutes later when her team wins the 2014 Tiwi Grand Final.

Welcome to the Tiwi Islands, two small islands about 80km north of Darwin, where AFL isn't just a passion but a way of life.

The Tiwis have the highest participation rate in AFL of any other community in Australia: about 900 of the Islands' 2600 population play, a staggering 35 per cent.

Big names in AFL have sprung from the Tiwi Islands, such as Hawthorn star Cyril Rioli, and Michael Long, who played for Essendon.

Their season runs during the hot, wet half of the year in the Top End, when hundreds of millimetres of rain can fall in a day but which makes surfaces easier to play on than the hard ground during the dry.

On Grand Final day the eight local teams have been whittled down to two: The Tuyu Buffaloes from Bathurst Island and the Mulwurri Magpies from Melville Island.

The Magpies put up a good fight but the Buffaloes claim the premiership, at 13 9 (87) to 12 5 (77), their second win since 2012.

Supporters flood the field, shouting and cheering as they mob the players, babies thrown up onto shoulders and older women weeping with pride as they join the fray.

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Tiwi Islands: where AFL is a way of life

Genetics clinic at Cancer Institute

by Dhaneshi Yatawara

A cancer genetics clinic will be conducted by the Human Genetics Unit of the University of Colombo at the Maharagama National Cancer Institute. The clinic will be held every Friday afternoon.

The purpose of the clinic is to identify individuals having hereditary cancer syndromes and provide thorough evaluation, genetic counselling and testing which will be beneficial for the patients and their families, according to Professor Vajira Dissanayake of the Human Genetics Unit, Colombo University.

"We will work with physicians and surgeons who treat the patients. The clinic is ready to provide service not only to cancer patients but also to their family members as well," Prof. Dissanayake said.

The cancer genetics clinic will conduct risk assessments for each patient for all forms of cancers and screening for early detection management, Genetic counselling and genetic testing.

"Members of families with records of cancer occurring in multiple generations or people with two or more close relatives having the same cancer can come to the clinic and get their risk assessments," he said.

And for those who underwent treatment for cancer that occurred in one section of paired organs of the body have the facility to check whether the cancer has spread to the other half of the organ. Several main genetic tests will be available at the clinic.

Tests to check effectiveness of the drugs on individual cancer patients medically known as pharmacogenomic tests, are also available at the clinic.

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Genetics clinic at Cancer Institute

Novel Gene-Finding Approach Yields a New Gene Linked to Key Heart Attack Risk Factor

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Newswise ANN ARBOR, Mich. Scientists have discovered a previously unrecognized gene variation that makes humans have healthier blood lipid levels and reduced risk of heart attacks -- a finding that opens the door to using this knowledge in testing or treatment of high cholesterol and other lipid disorders.

But even more significant is how they found the gene, which had been hiding in plain sight in previous hunts for genes that influence cardiovascular risk.

This region of DNA where it was found had been implicated as being important in controlling blood lipid levels in a report from several members of the same research team in 2008. But although this DNA region had many genes, none of them had any obvious link to blood lipid levels. The promise of an entirely new lipid-related gene took another six years and a new approach to find.

In a new paper in Nature Genetics, a team from the University of Michigan and the Norwegian University of Science and Technology report that they zeroed in on the gene in an entirely new way.

The team scanned the genetic information available from a biobank of thousands of Norwegians, focusing on variations in genes that change the way proteins function. Most of what they found turned out to be already known to affect cholesterol levels and other blood lipids.

But one gene, dubbed TM6SF2, wasnt on the radar at all. In a minority of the Norwegians who carried a particular change in the gene, blood lipid levels were much healthier and they had a lower rate of heart attack. And when the researchers boosted or suppressed the gene in mice, they saw the same effect on the animals blood lipid levels.

Cardiovascular disease presents such a huge impact on peoples lives that we should leave no stone unturned in the search for the genes that cause heart attack, says Cristen Willer, Ph.D., the senior author of the paper and an assistant professor of Internal Medicine, Human Genetics and Computational Medicine & Bioinformatics at the U-M Medical School.

While genetic studies that focused on common variations may explain as much as 30 percent of the genetic component of lipid disorders, we still dont know where the rest of the genetic risk comes from, Willer adds. This approach of focusing on protein-changing variation may help us zero in on new genes faster.

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Novel Gene-Finding Approach Yields a New Gene Linked to Key Heart Attack Risk Factor