Biology Lab Safety–Dissection and Microbiology – Flinn Scientific – Video


Biology Lab Safety--Dissection and Microbiology - Flinn Scientific
The biology lab can be hazardous too. Here are some proper procedures, instruments, and safe methods. To learn more about the Flinn Scientific Laboratory Safety Training Course visit us at: labsafety.flinnsci.comFrom:FlinnScientificViews:0 0ratingsTime:12:41More inEducation

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Biology Lab Safety--Dissection and Microbiology - Flinn Scientific - Video

Ethical Dilemma.wmv – Video


Ethical Dilemma.wmv
Two 10th Grade Biology are confronted with the Ethical Dilemma of Genetic Cloning. Stakeholders include those who have lost government, family members, infertility rights, therapeutic cloning, endangered species, biotech companies, and scientists.From:Jaime FordViews:0 0ratingsTime:09:28More inScience Technology

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Ethical Dilemma.wmv - Video

Faces of UVic Research: Alisdair Boraston – Video


Faces of UVic Research: Alisdair Boraston
Dr. Alisdair Boraston is a structural biologist and a professor at the University of Victoria. He is one of six Canadian scientists to be awarded a prestigious 2012 EWR Steacie Memorial Fellowship, he is currently a Michael Smith Foundation for Health Research Scholar and he is a Canada Research Chair in Molecular Interactions. He is one of the world #39;s leading experts on how carbohydrates and proteins interact in such processes as bacterial infection and the generation of bioethanol for fuels. In this video, he discusses these applications of his research and explains the basics of structural biology. Learn more at http://www.uvic.ca University of Victoria: http://www.uvic.ca University of Victoria #39;s Channel on YouTube: http://www.youtube.comFrom:FacesOfUVicResearchViews:2 0ratingsTime:01:22More inEducation

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Faces of UVic Research: Alisdair Boraston - Video

The Drug Bust — a 1971 Kent State University student film project. – Video


The Drug Bust -- a 1971 Kent State University student film project.
In 1971 I took a photography class. One assignment was to make a movie. Most people who were with me in university, including me, smoked marijuana from time to time. Eventually though, most of us got tired of it and, quit smoking. But for a few years it was great fun to get together with friends and smoke a joint. I read that marijuana was a "gateway" drug. Yes, it was, it opened a gate that showed us that the laws that made marijuana illegal were ridiculous. How nice that the country is slowly decriminalizing marijuana. This movie is dedicated to the memory of the man in the movie with the mustache, Dennis Seasock. Dennis was a great friend who, in the late 1970s, became a brilliant biology high school teacher in San Francisco. He was the life of the party. He was so lively that one night in 1978 when a crazy man decided to kill the happiest man standing in line waiting to see a movie, he chose Dennis. Dennis was stabbed to death. His killer got 25 years.From:oldmemories diehardViews:0 0ratingsTime:04:49More inPeople Blogs

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The Drug Bust -- a 1971 Kent State University student film project. - Video

Rainforest Destruction – 2012 PSA Video – Video


Rainforest Destruction - 2012 PSA Video
This one minute video explains how rainforest destruction and deforestation is evident and effects everyone. Rainforest destruction and deforestation is doing a lot of damage. However, we can change that. Multiple ways to help with this terrible tragedy. Watch the video for more information. Libby Yost, Jonah Blue, Miranda Mitchell, Hour 2 Honors Biology Class Piano by Libby Yost, playing "River Flows in You" by Yiruma. Shakopee Senior High School, Class of 2015.From:Libby YostViews:0 0ratingsTime:01:02More inEducation

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Rainforest Destruction - 2012 PSA Video - Video

Pomona Holds Memorial for Professor Gene Fowler

Kulsum Ebrahim

Students, friends and colleagues gathered in the SCC Hart Room on Wednesday to honor Professor Gene Fowler.

A memorial was held for Gene Fowler,a biology professor at Pomona College and one of the pioneers in establishing Pomona's environmental analysis program,in the Hart Room of the Smith Campus Center at Pomona on Wednesday. Fowler died Nov. 1 at Pomona Valley Hospital Medical Center after suffering from an illness.

Friends, colleagues and students remembered Fowler in an intimate setting. Speakers included Lenny Seligman, Chair of the Pomona Department of Biology; Pomona biology professors Nina Karnovsky and Clarissa Cheney; Nancy Hamlett, Visiting Professor of Biology at Harvey Mudd College; and Pomona mathematics professor Richard Elderkin.

E-mails sent by former students describing their favorite memories of Fowler were read aloud. Pomona biology professor Rachel Levin spoke for Alberto Sosa, a friend and colleague of Fowler from Argentina.

Playing in the background was a slideshow of photographs of Fowler with family members, students, friends and colleagues, as well as penguins, the subject of his research.

Crme brle was served at the memorial, asFowler was well known for caramelizing the sugar on crme brle with his chef's torch at Biology Department dinners.

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Pomona Holds Memorial for Professor Gene Fowler

Cancer drug shows promise in eradicating latent HIV infection

Public release date: 29-Nov-2012 [ | E-mail | Share ]

Contact: Cody Mooneyhan cmooneyhan@faseb.org 301-634-7104 Federation of American Societies for Experimental Biology

Bethesda, MDBreakthrough drugs have made it possible for people to live with HIV longer than ever before, but more work must be done to actually cure the disease. One of the challenges researchers face involves fully eradicating the virus when it is latent in the body. A new report appearing in the December 2012 issue of the Journal of Leukocyte Biology suggests that a cancer drug, called JQ1, may be useful in purging latent HIV infection by activating the virus in the presence of potent therapy essentially a dead end for the virus.

"This drug may be useful as adjunctive therapy in efforts to purge latent HIV reservoirs to eradicate infection," said Monty A. Montano, Ph.D., principal investigator from the Section of Infectious Diseases at Boston University Medical Campus, in Boston, Mass. "This drug functions synergistically with other HIV purging agents."

To make this discovery, scientists used cell lines that contained latent HIV, as well as cells from patients who were on potent antiretroviral therapy with no detectable virus in their blood. Researchers then added JQ1 to latently infected cells, at physiologic concentrations, and observed potent reactivation of latent HIV. The researchers also observed potent suppression of inflammatory genes in genome-wide expression studies using the same cells. JQ1 reactivation of latent HIV may make it vulnerable to current therapies.

"One thing that's been made clear by the decades of HIV research is that there is no magic bullet for curing this disease," said John Wherry, Ph.D., Deputy Editor of the Journal of Leukocyte Biology. "Rather, the progress that's been made has been a series of incremental steps that often build on other previously developed therapies. The hope is that the ability of JQ1 to make latent HIV 'visible' to other HIV drugs described in this report will be another cog in the gearwheel of an HIV cure."

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The Journal of Leukocyte Biology publishes peer-reviewed manuscripts on original investigations focusing on the cellular and molecular biology of leukocytes and on the origins, the developmental biology, biochemistry and functions of granulocytes, lymphocytes, mononuclear phagocytes and other cells involved in host defense and inflammation. The Journal of Leukocyte Biology is published by the Society for Leukocyte Biology.

Details: Camellia Banerjee, Nancie Archin, Daniel Michaels, Anna C. Belkina, Gerald V. Denis, James Bradner, Paola Sebastiani, David M. Margolis, and Monty Montano. BET bromodomain inhibition as a novel strategy for reactivation of HIV-1. J Leukoc Biol December 2012 92:1147-1154, doi:10.1189/jlb.0312165 ; http://www.jleukbio.org/content/92/6/1147.abstract

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Cancer drug shows promise in eradicating latent HIV infection

Fake biology teacher is jailed

29 November 2012 Last updated at 13:59 ET

A "pathological liar" who forged qualifications to get a job as a teacher has been jailed for 18 months.

Julia Rawlinson, 45, faked documents including a doctorate to get a biology teaching job at Westlands School in Torquay, Exeter Crown Court was told.

Rawlinson, of Brixham, Devon, admitted false representation and forgery.

Judge Philip Wassall told her: "You created layer upon layer of lies and blighted everyone you taught. There was not a single sign of regret."

The court heard Rawlinson's forged documents included a masters degree in science psychology and a doctorate of science psychology from Glasgow Caledonian University and a BSc in Biochemistry from the University of Witwatersrand in South Africa.

Staff at the school became suspicious and called the police.

Rawlinson had portrayed herself as chief examiner for A-level biology with Edexcel and had helped the school before summer 2011 with A-level biology project work.

She had been taken on as a temporary part-time staff member on 1 September of last year and her employment at Westlands was terminated early the next month.

Lee Bremridge, defending Rawlinson, said she was a "pathological liar who created a lie and came to believe it".

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Fake biology teacher is jailed

EWC professor recognized by scientific journal

Edward Waters College biology professor Prabir Mandals scientific review was the most downloaded within the Journal of Comparative Physiology B, according to the Springer Science+Business Media.

Mandal, a state-recognized researcher and environmental genotoxicity expert, is noted for his contributions of research and publications relating to health implications smoking has on African-Americans, specifically in inner cities.

His review, Dioxin: a review of its environmental effects and its aryl hydrocarbon receptor biology, describes the correlation between toxic chemicals, tumor and cancer development and its harmful effects on human DNA.

Although dioxins were found in pesticides, bleach, municipal and medical waste incinerators, Mandal confirms metal refining and iron sintering are leading causes of dioxins. Mandals review was first acknowledged as having been the most downloaded within the journal in 2005.

Mandal and Springer, the worlds leading scientific publisher and provider of the worlds largest eBook collection of Science, Technical and Medical media, recently agreed on a publishing deal for Mandal to publish a book on dioxin.

It is a tremendous honor and opportunity to be considered to author a book through an esteemed international publishing company, Mandal said. However, the best part is that our biology program will be impacted by the research that will occur in order to compile information for the publication and, eventually, the entire world will have access to the information.

Janay Cook: (904) 359-4310

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EWC professor recognized by scientific journal

SRU biology professor receives $50,000 grant

A SRU biology professor and University of Pittsburgh collaborators received a $50, 000 three-year research grant from the National Institute of Health to better understand the cause of health problems including Alzheimers disease and cystic fibrosis.

When Dr. Stacy Hrizo, a SRU biology professor, found out that her mother-in-law was diagnosed with dementia, she became interested in neurodegenerative research. According to the Institute for Dementia Research and Prevention, at least 5 million people in the U.S. have age-related dementias.

After Hrizo applied, she found Dr. Michael Palladino, a University of Pittsburgh pharmacology and chemical biology professor who accepted her as a post-doctoral researcher and developed this project, formally called, Protein Quality Control Mechanisms of Novel Soluble Substrates, with her. Hrizo and Palladino are specifically studying how sugar metabolism causes neurodegeneration, a disease that links to disorders like dementia.

Theres still a lot that we dont understand about the brain and about how it functions, Hrizo said. But the majority of the drugs that the industry uses target the process of the brain. Hrizo and researchers are studying the factors that affect this glycolytic disorder with fruit flies, a genetic model system called drosophila melanogaster, so they can study the flies from birth to death within about two months. (Dementia occurs at later stages from birth).

When you are studying mutations in a mouse or a human, it takes years before you see anything happening in the brain, Hrizo said.

Fruit flies are commonly used for biology research in genetics.

Dr. Andrew VanDemark, an assistant structural biology professor at Pitt, is another collaborator. He is creating crystals of the proteins to help the researchers study the structure changes in the mutations. Doctors have not found any certain treatment for neurodegenerative disorders.

We dont have very effective medicines against it, Hrizo said. But we do know that there are certain factors that affect your risk or probability of developing a neurodegenerative disorder where your brain cells die and get these symptoms. Some of these factors include diet and exercise. Doctors recommend dementia patients to maintain a healthy diet and exercise enough to slow the process of the symptoms.

Hrizo has been awarded $9, 718 for the first year and will receive a total of $48,406 by the last year. She is working on most of her study in the Pitts School of Medicine lab over the summer, and some is conducted at SRU. Hrizos goal is to screen 600 to 800 different genes within the next two years and then confirm their findings in the third year. Hrizo and collaborators project is a type of cell biology research which is under the same category as, Robert Lefkowitz and Brian Kobilka, the 2012 Nobel Prize winners in chemistry. They won the $1 million prize for identifying a class of receptors in the brain and understanding how they work.

The National Institute of Health is funding the researchers to look at how cells are destroying enzymes that are functioning and folded properly in sugar metabolism. This disease, called TPI deficiency, starts to take a negative effect on the brain. Hrizos experiment is on fruit flies because she can control the lighting, temperature, how much and when to feed them in the lab. Palladino is putting the mutations in the fruit flies to compare how different mutations cause diseases. He will look at the progression of each disease in a population of genetically identical fruit flies which lasts in a course of about two months.

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SRU biology professor receives $50,000 grant

Biology behind brain development disorder: Mutations in the gene, UBE3B, cause a rare genetic disorder in children

ScienceDaily (Nov. 29, 2012) This study describes how a combination of sequencing and mouse models were used to identify the gene responsible for a brain developmental disorder seen in four patients. The study also shows that the biology uncovered in the mouse model helps to understand the symptoms in patients.

The work highlights how animal models are a complementary approach for determining the causal genes and for understanding the biology behind genetic disorders.

Researchers have defined the gene responsible for a rare developmental disorder in children. The team showed that rare variation in a gene involved in brain development causes the disorder. This is the first time that this gene, UBE3B, has been linked to a disease.

By using a combination of research in mice and sequencing the DNA of four patients with the disorder, the team showed that disruption of this gene causes symptoms including brain abnormalities and reduced growth, highlighting the power of mouse models for understanding the biology behind rare diseases.

"Ubiquitination, the biological pathway UBE3B is involved in, is crucial in neurodevelopment," says Dr Guntram Borck, lead author from the University of Ulm. "We have studied several patients with this rare condition, and by sequencing the coding regions of the genome of these patients we found mutations implicating the gene UBE3B. This result was confirmed by studies performed in mice by our collaborators at the Sanger Institute.

At the Sanger researchers deleted the gene in mice and found that they had symptoms that were quite similar to those in the patients with UBE3B mutations including; reduced body weight and size, and reduced size of the brain.

The studies in mice also uncovered other defects underlying the disorder. Mice with the gene deletion had reduced cholesterol levels, a symptom that was seen by the team in three of the patients. This observation suggests that a defect in cholesterol metabolism is associated with this syndrome.

"Both techniques, DNA sequencing and deleting the gene in mice, support the finding that disruption of UBE3B causes this syndrome," says Dr David Adams, lead author from the Wellcome Trust Sanger Institute. "We can now learn much more about this syndrome by studying these mice. They also represent a pre-clinical model in which we may trial potential new therapies.

"This is the first time that this gene has been implicated in any disorder."

DNA sequencing has greatly improved the identification of variants associated with developmental disorders. But the challenge still remains for researchers to identify which of these variants, there are usually several hundred identified in each patient, cause the disorder. Animal models are a complementary approach for determining the causal gene and for understanding the biology behind genetic disorders..

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Biology behind brain development disorder: Mutations in the gene, UBE3B, cause a rare genetic disorder in children

Biology behind brain development disorder

Public release date: 29-Nov-2012 [ | E-mail | Share ]

Contact: Aileen Sheehy press.office@sanger.ac.uk 0044-012-234-96928 Wellcome Trust Sanger Institute

Researchers have defined the gene responsible for a rare developmental disorder in children. The team showed that rare variation in a gene involved in brain development causes the disorder. This is the first time that this gene, UBE3B, has been linked to a disease.

By using a combination of research in mice and sequencing the DNA of four patients with the disorder, the team showed that disruption of this gene causes symptoms including brain abnormalities and reduced growth, highlighting the power of mouse models for understanding the biology behind rare diseases.

"Ubiquitination, the biological pathway UBE3B is involved in, is crucial in neurodevelopment," says Dr Guntram Borck, lead author from the University of Ulm. "We have studied several patients with this rare condition, and by sequencing the coding regions of the genome of these patients we found mutations implicating the gene UBE3B. This result was confirmed by studies performed in mice by our collaborators at the Sanger Institute.

At the Sanger researchers deleted the gene in mice and found that they had symptoms that were quite similar to those in the patients with UBE3B mutations including; reduced body weight and size, and reduced size of the brain.

The studies in mice also uncovered other defects underlying the disorder. Mice with the gene deletion had reduced cholesterol levels, a symptom that was seen by the team in three of the patients. This observation suggests that a defect in cholesterol metabolism is associated with this syndrome.

"Both techniques, DNA sequencing and deleting the gene in mice, support the finding that disruption of UBE3B causes this syndrome," says Dr David Adams, lead author from the Wellcome Trust Sanger Institute. "We can now learn much more about this syndrome by studying these mice. They also represent a pre-clinical model in which we may trial potential new therapies.

"This is the first time that this gene has been implicated in any disorder."

DNA sequencing has greatly improved the identification of variants associated with developmental disorders. But the challenge still remains for researchers to identify which of these variants, there are usually several hundred identified in each patient, cause the disorder. Animal models are a complementary approach for determining the causal gene and for understanding the biology behind genetic disorders..

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Biology behind brain development disorder

Yu-Gi-Oh – Video


Yu-Gi-Oh
On my way to theatre rehearsal, I saw a nice-looking moon in a small gap between the clouds. It made me make a vlog, in which I speak about experiments for biology class. Yu-Gi-Oh Burst Chapter 2 is also being worked on.From:Auke1993Views:0 0ratingsTime:02:47More inPeople Blogs

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Yu-Gi-Oh - Video

Bach Concerto No 1 in C minor for Two Pianos Mvt 2 – Video


Bach Concerto No 1 in C minor for Two Pianos Mvt 2
The Orchestra of Southern Utah welcomed featured artists Gerald Rheault and Erika Dobson at the Bach and Bruckner Concert. The artists performed Bach #39;s Concerto No. 1 in C minor for 2 Pianos. Rheault is the Utah Shakespeare Festival #39;s Greenshow Music Director and Associate Conductor. He plays keyboard in the Les Miserables pit orchestra. He serves as Assistant Conductor for OSU. Rheault also currently holds the position of SUU College of Visual and Performing Arts Staff Music Director, Conductor and Accompanist. Dobson is a student at SUU majoring in biology and minoring in music. She has performed with the National Federation of Music Club for several years and was selected to perform at Utah State Solo Ensemble. In addition to playing the piano, Dobson is also proficient with the flute. Johann Sebastian Bach was a German composer, organist, harpsichordist, violist, and violinist. His abilities as an organist were highly respected throughout Europe during his lifetime, although he was not widely recognized as a great composer until the early 1800s. He is now generally regarded as one of the greatest composers of all time. Filmed by Steven Swift. Copyright 2012 Orchestra of Southern Utah. All Rights Reserved.From:Sara PennyViews:2 0ratingsTime:05:42More inMusic

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Bach Concerto No 1 in C minor for Two Pianos Mvt 2 - Video