Category Archives: Transhuman News

David Icke: Human Genetics, The Religion of Death
Alex talks with author and presenter David Icke about election fraud and human genetics. http://www.davidicke.com ORDER THIS SURVIVAL DVD AND START GETTING PREPARED TODAY AT... http://www.infowarsshop.com Joel Skousen sits down with Alex Jones and discusses strategies on how to prepare for and survive major disasters. http://www.infowars.com http://www.prisonplanet.tv twitter.com http://www.facebook.com [Join The Alex Jones Team and Start Getting Healthy Today!] http://www.infowarsteam.com Get all your Youngevity Products such as Beyond Tangy Tangerine, the Alex Pack and Pollen Burst. These supplements are a great way to get your essentials vitamins, minerals, amino acids, and other beneficial nutrients [[[ProPur Water Filtration]]] http://www.infowarsshop.comFrom:TheAlexJonesChannelViews:12071 298ratingsTime:58:00More inPeople Blogs

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David Icke: Human Genetics, The Religion of Death - Video

Alex Jones - David Icke: Human Genetics, The Religion Of Death
Alex Jones talks with author and presenter David Icke about election fraud and human genetics. ___________________________________________ http://www.infowars.com http http://www.infowars.net http http://www.youtube.com twitter.com http://www.facebook.com _______________________________________ http://www.davidicke.com http://www.youtube.comFrom:Mohammed AminViews:1 0ratingsTime:58:00More inNonprofits Activism

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Alex Jones - David Icke: Human Genetics, The Religion Of Death - Video

David Icke Exposes ROYAL PAEDOPHILES
Alex talks with author and presenter David Icke about election fraud and human genetics. .From:sswemmerViews:21 2ratingsTime:58:00More inEducation

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David Icke Exposes ROYAL PAEDOPHILES - Video

ALEX JONES Talks To DAVID ICKE: Human Genetics, Election FRAUD The Religion Of DEATH
ALEX JONES Talks To DAVID ICKE: Human Genetics, Election FRAUD The Religion Of DEATH Alex talks with author and presenter David Icke about election fraud and human genetics. TAGS: David Icke Alex Jones New World Order NWO Illuminati Conspiracy Truth Bush Government Propaganda Brain Washing WW3 wwiii Globalist Elite Takeover Gerald Celente Max Keiser Financial Economic Crisis Freedom Economy Tyranny 9/11 911 FBI CIA Zion Zionism Zionist Rothschild Royal Family Tsiyonuttimes 2012 2013 truth wake up matrix system plan agenda life government control cash trends bilderberg secret rich trendy fear divide rule religion income sheeple America US USA uk britain war ww3 terror false flag attack evil economy crisis gold silver investment bullion banker alex jones infowars gerald celente lindsey williams rothschild Tsiyonuttimes 2012 2013 US USA America Paul Ryan Barack Michelle Obama Mitt Romney Joe Biden President Vice President Election Votes Voters Polls Speech Debate Democrat Republican Tea Party George Bush Bill Hilary Clinton Forward Change 47% Middle Class Income Policy PoliticsFrom:Tsiyonut timesViews:0 0ratingsTime:58:00More inNews Politics

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ALEX JONES Talks To DAVID ICKE: Human Genetics, Election FRAUD

SAN JOSE, Calif., Nov. 5, 2012 /PRNewswire/ -- Ariosa Diagnostics today announced that it will present data from two recent studies on non-invasive prenatal testing (NIPT) at the American Society of Human Genetics (ASHG) Annual Meeting at The Moscone Center in San Francisco, Calif. from Nov. 6 - 11, 2012. The studies highlight a directed cell-free (cfDNA) analysis method of NIPT.

(Logo: http://photos.prnewswire.com/prnh/20121105/NE05794LOGO)

A poster titled "Non-invasive Chromosomal Evaluation (NICE) Study: Results of a Multicenter, Prospective, Cohort Study for Detection of Fetal Trisomy 21 and Trisomy 18," examines the performance of Ariosa's Harmony Prenatal Test, a directed cfDNA analysis method of detecting fetal trisomies. The NICE study represents the largest cohort study in NIPT. Pregnant women at gestational age 10 weeks or later were enrolled from 50 clinical sites in the U.S. and Europe. The poster will be presented on Thursday, Nov. 8, from 3:15 - 4:15 p.m. PST in The Moscone Center Exhibit Hall, Lower Level South.

A second poster titled "The Fetal Fraction of cell-free DNA in Maternal Plasma is Not Affected by a priori Risk of Fetal Trisomy," examines possible clinical factors that might influence the fetal fraction of cfDNA in maternal plasma. The amount of fetal cfDNA in maternal plasma is a major determinant of the ability to detect trisomy. A comparative analysis shows no difference in fetal cfDNA levels between high- and low-risk pregnant women providing support that NIPT performance will be just as robust in a general pregnancy population. This poster will be presented on Thursday, Nov. 8, from 3:15 - 4:15 p.m. PST in The Moscone Center Exhibit Hall, Lower Level South.

For more information about Ariosa and the company's directed cfDNA analysis method of NIPT, please visit us at Booth #1609.

About Ariosa Diagnostics

Ariosa Diagnostics, Inc., is a molecular diagnostics company committed to innovating together to improve patient care. The flagship product, the Harmony Prenatal Test, is a safe, highly accurate and affordable prenatal test for maternal and fetal health. Led by an experienced team, Ariosa is using its proprietary technology to perform a directed analysis of cell-free DNA in blood. The Harmony test equips pregnant women and their healthcare providers with reliable information to make decisions regarding their health, without creating unnecessary stress or anxiety.

The company began operations in 2010 and is headquartered in San Jose, Calif. For more information, visitwww.ariosadx.com.

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Ariosa Diagnostics to Present Clinical Data on the Harmony™ Prenatal Test at American Society of Human Genetics Annual ...

Public release date: 6-Nov-2012 [ | E-mail | Share ]

Contact: Cathy Yarbrough press@ashg.org 858-243-1814 American Society of Human Genetics

Scientists have adopted a novel laboratory approach for determining the effect of genetic variation on the efficiency of the biological process that translates a gene's DNA sequence into a protein, such as hemoglobin, according to a presentation, Nov. 6, at the American Society of Human Genetics 2012 meeting in San Francisco.

In the 0.1% of the DNA that differs between any two individuals, scientists search for the biological mechanisms underlying human genetic differences, including disease susceptibility.

"How exactly these slight changes in the DNA affect the biology of the human body is not known in most cases," said Constantin Polychronakos, M.D., professor of pediatrics, experimental medicine and human genetics at McGill University, Montreal, Canada.

"We decided to investigate the possibility that some of these changes may alter the translation of RNA into protein, a question that had not been systematically examined before," he added.

Translation is the final stage of gene expression at which the gene's DNA recipe for a protein can be modified, said McGill University scientist Quan Li, Ph.D., who presented the research.

In general, genomic studies have focused on finding links between diseases and variation in DNA. However, the new study takes a big step toward understanding how that variation affects the production of proteins, which are the molecules that most directly affect health and disease.

The study was designed to determine the effect of single-nucleotide polymorphisms (SNPs), which are variations in the DNA sequence, on the process of translation, Dr. Li said.

Translation begins when a gene's DNA sequence is transcribed into the messenger RNA (mRNA) molecule that carries the transcript, or the blueprint for the protein encoded by the gene, to ribosomes, where proteins are manufactured in a cell.

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New method helps link genomic variation to protein production

Public release date: 6-Nov-2012 [ | E-mail | Share ]

Contact: Cathy Yarbrough press@ashg.org 858-243-1814 American Society of Human Genetics

Humans share over 90% of their DNA with their primate cousins. The expression or activity patterns of genes differ across species in ways that help explain each species' distinct biology and behavior.

DNA factors that contribute to the differences were described on Nov. 6 at the American Society of Human Genetics 2012 meeting in a presentation by Yoav Gilad, Ph.D., associate professor of human genetics at the University of Chicago.

Dr. Gilad reported that up to 40% of the differences in the expression or activity patterns of genes between humans, chimpanzees and rhesus monkeys can be explained by regulatory mechanisms that determine whether and how a gene's recipe for a protein is transcribed to the RNA molecule that carries the recipe instructions to the sites in cells where proteins are manufactured.

In addition to improving scientific understanding of the uniqueness of humans, studies such as the investigation conducted by Dr. Gilad and colleagues could have relevance to human health and disease.

"Through inter-species' comparisons at the DNA sequence and expression levels, we hope to identify the genetic basis of human specific traits and in particular the genetic variations underlying the higher susceptibility to certain diseases such as malaria and cancer in humans than in non-human primates," said Dr. Gilad.

Dr. Gilad and his colleagues studied gene expression in lymphoblastoid cell lines, laboratory cultures of immortalized white blood cells, from eight humans, eight chimpanzees and eight rhesus monkeys.

They found that the distinct gene expression patterns of the three species can be explained by corresponding changes in genetic and epigenetic regulatory mechanisms that determine when and how a gene's DNA code is transcribed to a messenger RNA (mRNA) molecule.

Dr. Gilad also determined that the epigenetics process known as histone modification also differs in the three species. The presence of histone marks during gene transcription indicates that the process is being prevented or modified.

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Humans, chimpanzees and monkeys share DNA but not gene regulatory mechanisms

Public release date: 6-Nov-2012 [ | E-mail | Share ]

Contact: Cathy Yarbrough press@ashg.org 858-243-1814 American Society of Human Genetics

A multi-institutional team of researchers has sequenced the DNA of 6,700 exomes, the portion of the genome that contains protein-coding genes, as part of the National Heart, Lung and Blood Institute (NHLBI)-funded Exome Sequencing Project, one of the largest medical sequencing studies ever undertaken.

Scientists participating in the project initially expected that individual rare variants would have a greater effect on over 80 heart, lung and blood related traits and diseases of high public health significance, said Suzanne M. Leal, Ph.D., professor and director, Center for Statistical Genetics in the Department of Molecular and Human Genetics of Baylor College of Medicine in Houston, TX.

The researchers found that many (1.1 million) of the 1.2 million coding variants that they identified in exome data from 4,420 European-Americans and 2,312 African-Americans occurred very infrequently in the population and often were only observed in a single individual, explained Dr. Leal, who presented the findings today at the American Society of Human Genetics 2012 meeting.

Dr. Leal added that most of the observed coding variants are population specific, occurring in either European or African Americans. "Of the identified variants, about 720,000 change the genetic code in a manner that could produce flawed proteins. Yet the role played by most of these variants in disease development has not been established," she said.

The major goal of the project was to understand how variation in the exome affects heart, lung and blood related traits and diseases.

The study participants were selected from a sample of over 220,000 individuals who participated in another National Institute of Health (NIH) supported study that had collected extensive medical data on the participants. "Individuals were selected to have a disease endpoint of interest or an extreme trait value of public health importance," said Dr. Leal.

By sequencing the exomes of 91 cystic fibrosis patients, Dr. Leal and her research colleagues discovered and replicated an association between variants in the DCTN4 gene and when a patient first develops a Pseudomonas aeruginosa airway infection.*

The researchers were also able to replicate many known associations between individual DNA variants and traits, such as high blood levels of low-density lipoprotein, known as the 'bad' cholesterol, and C-reactive protein, which increases the body's response to inflammation.

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Surprising findings from NHLBI Exome Sequencing Project reported