Daily Archives: July 5, 2017

Scientists are questioning the idea that the human lifespan has a limit – Yahoo News

Posted: July 5, 2017 at 10:47 pm

An elderly man swims

(Al Bello/Getty Images)

Jeanne Calment, the French woman who holds the record for the longest verified lifespan, died in 1997 at 122 years old.

Few people, of course, everbecome supercentenarians 110 years old or older and even fewer hit 115.

So few people have exceeded that age, in fact, that a group of researchers published an analysis in the journal Nature last year arguing that the human species' lifespan plateaus around115.

But a number of scientists are now rebutting that analysis with five separate commentaries published in Nature on June 28.

The authors of these piecesargue that the original analysis relied onstatistics that were incomplete or analyzed in a way that led to a falseconclusion. They suggesttwo alternatives: We either don't have enough data to know if the human lifespan has a limit, or theplateau is closer to125 than 115.

"The available data are limited, there aren't that many supercentenarians," Maarten Pieter Rozing, a professor at the University of Copenhagenwho co-authoredone commentary, told The Scientist. "And I think there are no strong arguments that show there is a decline [in the rate at whichlifespans areincreasing]."

Life expectancy has crept up fairly steadily over the past 150 years or so. But Xiao Dong, Brandon Milholland, and Jan Vijg, the authors of the original analysis, argue that comparing the life expectancy of supercentenarians to theage at whichthey died can reveal thenatural limit ofthe human lifespan.

(iStock)

The scientists used data on maximum reported age at death split into two sets based on supercentenarians from the US, UK, Japan, and France. Thefirst set covered deathsfrom 1968 to 1994 a period when the maximum age was inchingup. But bythe time covered in the next dataset, from 1995 to 2006, the ageseemed to plateau or even slightly be on the decline (exceptions like Calment aside).

Life expectancy, however, rose throughout both time periods.The scientists therefore concludedthatbecause humans' maximum age didn't keeprising with life expectancy, it appeared a limit had been reached.

Even if we were to cure various diseases like cancer or Alzheimer's, those scientists still claimed thathumans would probablybe unlikely to live past 115. And they put thechances ofa person live past 125 atless than 1 in 10,000.

elderly chinese couple

(David Gray/Reuters)

The authors of the recent rebuttals say that because there are so few supercentenarians out there, the number of deaths for this age group between 1995 and 2006 is too small to yield reliableconclusions. There just haven't been enough supercentenarians to really pinpoint amaximum age.

As people live longer, it's likely that more will push past that supposed limit, the authors of the rebuttals argue it'll just take time toget there.

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"[T]he idea of a set limit to human longevity is not strongly supported by what is being discovered about the biology of ageing," Rozing and his co-authors wrote in their commentary. "The continuing increase in human life expectancy that has occurred over recent decades was unforeseen. It provides evidence for greater malleability of human ageing than was originally thought."

Over the span of human history, many of the lifespan increases we've seen would have been unimaginable at some point. Thoseliving200 years ago, for example, would have thought it wascrazy that peoplecould regularly liveto be80. Yet here we are.

Rozing told The Scientistthat there'san easy way to find out whose hypothesis is correctabout the maximum lifespan.

"[W]e can just wait and see who's right," he said.

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How To Treat Eczema on Your Face – Men’s Health

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How To Treat Eczema on Your Face
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As someone who has struggled with facial eczema since childhood, I can say that dealing with it, let alone treating it, can be hell. It wasn't until I started to notice the hallmark symptomsitchy red, scaly patches that I realized my generic ...

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Dr. Cannaday’s eczema cure, part 2 – Sedalia Democrat

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Sedalia dermatologist Dr. Julian Cannaday achieved fame and wealth treating eczema, a skin disease marked by rough, itchy, scaly patches on the skin. In the book he wrote advocating his treatment for eczema, he questioned the currently available treatments and the doctors who prescribed them. He also offered his own cure.

Cannaday proposed that eczema was difficult to cure because most physicians treated the effects and ignored the cause. He believed he knew the cause, which he claimed was the buildup of acid in the blood and the inability of the body to cleanse the blood, forcing the acid to be excreted through the skin, which developed itchy patches where the acid seeped out. His explanation, and the certainty with which he pronounced it, is interesting, in that the exact cause of eczema remains unknown.

The website of the prestigious Mayo Clinic suggests several contributing factors, including dry irritable skin caused by too much exposure to hot or cold water or harsh cleaning agents, allergies to dyes or perfumes in soaps or lotions, and environmental conditions such as exposure to hot dry air. Cannaday explains that these factors may aggravate eczema by irritating skin already damaged by the acid excreted through the skin.

Some treatments offered during the early 20th century tried to kill germs that were found in the scabs that developed on the skin. Cannaday believed the germs were carried by the fingers of the patient who scratched the intensely itching skin and were not the cause of the disease. In addition, the things prescribed to treat the skintar, zinc, carbolic acid, corrosive sublimates [used] to kill the germs were so strong they injured the skin

Cannaday dismissed some of the treatments proposed by other doctors, such as cod liver oil, though a deficiency of Vitamin D, a substance found in cod liver oil, may contribute to eczema. Cannaday also dismissed ointments prescribed by other doctors.

Cannadays treatment consisted of the use of a diet that avoided foods that build up acid during digestion. His prescribed a special diet that he said would allow the patient to eat enough food to keep hunger away while not building up acid.

The second part of Cannadays treatment was a blood tonic taken as either pills or liquid form three times per day. He believed the tonic would improve digestion.

The third part of his treatment was the use of a special wash for the affected skin.

Cannadays book does not list the foods allowed on his diet, nor does it give any hints as to the composition of the tonic or the wash. For the treatment, one had to pay $12.50 ($308 in todays purchasing power) for two months worth of tonic, powder to mix with water to make the wash, and a written copy of the diet.

We still do not know the exact nature of Cannadays cure. Perhaps someday, someone will find in the attic a copy of the diet, a tonic pill or two, and some of the wash power so they can be analyzed for their content.

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Hopkins joins gene cloning project to advance medicine development – Baltimore Sun

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Scientists at Johns Hopkins, Rutgers and Harvard universities, as well as the University of Trento in Italy, have created a new technique that allows thousands of genes in a DNA sequence to be cloned at once.

Researchers hope the advance in gene cloning will allow them to more quickly identify markers for diseases and discover new medicines.

Until now genes had to be cloned individually in a time-consuming process. The new molecular method allows thousands of the long DNA strands that make up genes to be isolated and cloned at the same time.

The discovery was published June 26 in Nature Biomedical Engineering.

"Our goal is to make it cheap and easy for any researcher in any field to clone and express the entire set of proteins from any organism," said Ben Larman, an assistant professor of pathology in the Johns Hopkins School of Medicine and the study's co-senior author, in a statement. "Until now, such a prospect was only realistic for high-powered research consortia studying model organisms like fruit flies or mice."

The scientists call their technique for capturing DNA strands that make up genes the LASSO method, for long adapter single-stranded oligonucleotid. They also liken it to capturing cattle with a rope.

The new process speeds up the genes' creation of proteins, which manage cell activity, compared to the old process of cloning individual genes.

To test the method, the scientists sought to capture more than 3,000 DNA strands from the E. coli bacterial genome, commonly used as a model organism, and were successful with most of the targets. They also were able to use the strands to analyze what the gene's proteins do.

"We're very excited about all the potential applications for LASSO cloning," Larman said. "Our hope is that by greatly expanding the number of proteins that can be expressed and screened in parallel, the road to interesting biology and new therapeutic biomolecules will be dramatically shortened for many researchers."

The next step, already underway, is improving the cloning process and building libraries of proteins from DNA samples for use in research, said Biju Parekkadan, an associate professor in the Department of Biomedical Engineering at Rutgers University-New Brunswick.

Funding for the research came from the Shriners Hospitals for Children, the Prostate Cancer Foundation and the National Institutes of Health.

Larman, Parekkadan and a Harvard scientist on the project have sought a patent for the method, which is pending.

meredith.cohn@baltsun.com

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With powerful new technique, scientists can clone thousands of genes at once – The Hub at Johns Hopkins

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By Chanapa Tantibanchachai

Scientists at Johns Hopkins, Rutgers, the University of Trento in Italy, and Harvard Medical School report they have developed a new molecular technique that can be used to isolate thousands of long DNA sequences at the same time, more than ever before possible.

According to the researchers, the new technologyknown as LASSO cloningspeeds up the creation of proteins, the final products of genes, and is likely to lead to far more rapid discovery of new medicines and biomarkers for scores of diseases.

Historically, figuring out what a gene does by cloning its DNA and expressing its protein was done one gene at a time. The new technology simultaneously clones and expresses thousands of protein-coding DNA sequences in a single reaction.

In a report on the technique's development, published online June 26 in Nature Biomedical Engineering, the researchers describe their novel molecular approach to simultaneously clone and express thousands of protein-coding DNA sequences in a single reaction. Historically, figuring out what a gene does by cloning its DNA and expressing its protein was done one gene at a time.

"Our goal is to make it cheap and easy for any researcher in any field to clone and express the entire set of proteins from any organism," says Ben Larman, an assistant professor of pathology at the Johns Hopkins University School of Medicine and the study's co-senior author. "Until now, such a prospect was only realistic for high-powered research consortia studying model organisms like fruit flies or mice."

The new paper describes a new type of captured DNA strand, a tool the authors refer to as a LASSO probe; LASSO stands for long adapter single-stranded oligonucleotide. Collections of these LASSO probes can be used to grab desired DNA sequencesmuch like a rope lasso is used to capture cattlebut in this case thousands at a time in a single effort.

Each target gene sequence can be up to a few thousand DNA base pairs long, which is the typical size of a gene's protein-coding sequence. The new technique is an improvement on an older method called molecular inversion probes, which is able to capture only about 200 bases of DNA, Larman says.

In a proof-of-concept study, LASSO probes were used to simultaneously capture more than 3,000 DNA fragments from the E. coli bacterial genome. The team successfully captured at least 75 percent of the gene targets. Importantly, the researchers say, these sequences are captured in a way that permits scientists to analyze what the genes' proteins do, as demonstrated by conferring antibiotic resistance to an otherwise susceptible cell.

"We're very excited about all the potential applications for LASSO cloning," Larman says. "Our hope is that by greatly expanding the number of proteins that can be expressed and screened in parallel, the road to interesting biology and new therapeutic biomolecules will be dramatically shortened for many researchers."

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Asthma drug shows promise in treating obesity and diabetes – University of Michigan Health System News (press release)

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ANN ARBOR, Mich. -- After 12 weeks of taking an anti-inflammatory asthma drug, obese patients with type 2 diabetes showed a clinically significant drop in blood glucose.

The drug amlexanox, prescribed in Japan to treat asthma, appeared to free the metabolic system to burn more energy. A subset of patients had improved fatty liver disease and insulin sensitivity, a response seen among those who started the clinical trial with higher levels of inflammation in their fat tissue than others.

While the discovery at Michigan Medicine and the University of California at San Diego is not ready for the clinic, it reveals an inflammatory link between obesity and type 2 diabetes.

Inflammation is the bodys natural response to injury and illness, but chronic inflammation caused by obesity is believed to promote insulin resistance, a main feature of diabetes.

We are beginning to understand the role this form of internal inflammation plays in the development of chronic diseases like diabetes, says lead study author Elif Oral, M.D., director of the MEND Obesity and Metabolic Disorder Program at Michigan Medicine. Ultimately we may be able to personalize therapy based on the degree of inflammation present at baseline which is a new concept.

Oral is an endocrinologist and translational scientist at Michigan Medicine, the University of Michigans academic medical center where the clinical trial was conducted and analyzed.

Tissue analysis was led by study author Alan R. Saltiel, Ph.D., at U-C San Diego, along with scientists at the Salk Institute for Biological Sciences.

In the Cell Metabolism study, researchers identified a molecular signature in obese patients with type 2 diabetes who responded to the drug amlexanox.

When we looked at the drug-treated group we saw a bimodal distribution, that is, there were some responders and some nonresponders. We didnt understand why, so we did a molecular analysis from biopsies of fat cells we took from patients at the beginning and end of the study, says Saltiel, director of the Institute for Diabetes and Metabolic Health at U-C San Diego

In the responder group, the level of inflammation in fat was higher than in the nonresponder group at the beginning of the study, indicating that there is something about inflammation that predisposes a person to respond. And, what was really amazing was that there were more than 1,100 gene changes that occurred exclusively in the responders.

The glucose-lowering effects of amlexanox were first discovered in mice during research at the University of Michigan where Saltiel served as director of the Life Sciences Institute at the U-M.

Promising results

Amlexanox is an inhibitor of two enzymes, IKK and TBK1. In previous studies, Saltiel and U-M researchers discovered that these two enzymes are induced in obese mice, causing a drop in energy expenditure or reduction in calories burned.

This prompted them to look for inhibitors of these enzymes by screening a library of 150,000 chemicals. They found amlexanox. Giving obese mice the inhibitor caused them to lose weight, while their sensitivity to insulin increased, improving their diabetes and fatty liver disease.

The human trial revealed that gene changes that occurred in the mouse model also happened in the human responder group. Blood sugar in the clinical trial patients went down as genes involved in the expenditure of energy changed.

The proof of concept trial began with an unblinded safety trial of six patients. It was followed by a controlled trial of 42 obese patients with type 2 diabetes.

Half of the patients were randomized to a placebo group while the other half received amlexanox for three months. Blood sugar, insulin sensitivity, weight and liver fat were measured. A biopsy of fat cells from each patients midsection was taken before and after the trial to measure changes in gene expression.

The most exciting part of this is that we have a new drug that has never been studied before, says Saltiel. Its a new mechanism for a diabetes and fatty liver drug. Its promising, but there are a lot of questions that need to be answered still.

Among them: Which gene changes are the most important to target? Whats the right drug dosage? What time of day should it be administered? How often should patients take the drug? Can the percentage of responders be increased? Will the beneficial effects of the drug be sustained for a longer time?

One-third of the participants in the blinded study responded. Among responders with nonalcoholic fatty liver disease, an improvement was also seen.

Researhers are planning follow-up trialsto look at whether its possible tostratify patients who are likely to respond based on the degree of underlying inflammation, and explore if other drug combinations can be used with amlexanox.

We are grateful for patient participation and hope that our patients will respond with the same enthusiasm to our new trials. Without patients volunteering, the sort of study can never happen, says Oral.

Primary support for the research came from the National Institutes of Health High Risk High Reward grant R21DK098776.

Additional authors include Shannon M. Reilly, Andrew V. Gomez, Rasimcan Meral, Laura Butz, Nevin Ajluni, Thomas C. Chenevert, Evgenia Korytnaya, Adam H. Neidert, Rita Hench, Diana Rus, Jeff Horowitz, BreAnne Poirier, Peng Zhao, Kim Lehmann, Mohit Jain, Ruth Yu, Christopher Liddle, Maryam Ahmadian, Michael Downes and Ronald M. Evans.

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Make uncivil speech socially unacceptable | Editorials | mtexpress.com – Idaho Mountain Express and Guide

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Baby boomers once came into adulthood fighting for the freedom to say crude words on television. As they reach the ends of their careers in this 21st century, they should recognize that the pendulum has swung too far. They should demand a return to civil speech.

Political correctness is a catch-all term for using self-restraint when talking. It means restraining derogatory comments about race, gender or creed aimed at individuals or groups of people. It means restraining fighting words.

Engagement in political correctness does not mean that people may not protest or vociferously disagree. It means that insults, profanity or violence used to deny the rights of others in this free nation are not acceptable behaviors.

On the fringe left this year, groups dressed in black with covered faces and calling themselves anti-fa, short for anti-fascists, broke windows and defaced buildings in supposed opposition to a politically incorrect speech on the University of California-Berkeley campus. It is not at all certain they were students, but that is relevant only in that the criminal property destruction added to the universitys unfair reputation for harboring antisocial activists.

On the fringe right, a white supremacist hurled racist slurs at two young women on a train in Portland, Ore., killed two men who came to their defense, then ranted in a courtroom that being politically correct is an affront to freedom.

Since the nations founding, Americans have seen many changes in what is considered socially acceptable speech. Changes in speech create social norms that can prevent, cool or fuel incendiary confrontations. For capturing hearts and minds, social norms are often more effective than laws.

For example, cigarette smoking was once ubiquitous despite scientific proof of disastrous health consequences. Yet, it wasnt until smoking became socially unacceptable that the numbers of smokers began to fall. Ashtrays that once were everywhere barely exist today.

Political speech, delivered over the air or in the streets, is a precious right that includes the distasteful, the ignorant, the politically incorrect. However, just because citizens can use uncivil speech doesnt mean we should.

When uncivil dialogue becomes political partisanship and then hardens into armed conflict, the times demand a return to the lessons of childhood: Speak your mind, but dont hurl profane personal insults. Do unto others as you would have them do unto you. Think before you speak.

This will make America a better, stronger and smarter place.

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North Korea’s missile test gives Trump his biggest challenge – CNN

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The North Korean dictator's first successful intercontinental ballistic missile test on Monday did more than shake up strategic calculations in the Pacific.

It presented Trump with his first real test on the global stage as he flies off to meet far more experienced leaders at the G20 summit in Germany, some of whom are ill-disposed to help him and don't have the US's best interests at heart.

It's a trip that will now be judged on Trump's capacity to secure not just international condemnation of North Korea's actions, but to advance US efforts to change the strategic calculation in Pyongyang.

The mission will test Trump's skill at wielding US power, building international coalitions behind American foreign policy goals and framing innovative policy approaches that haven't yet been tried and that don't fit neatly into the "America First" doctrine that is driving his foreign policy.

Forget the tweetstorms, slams at "fake news" journalists and morale boosting rallies before crowds who thrill to Trump's politically incorrect rhetorical blasts.

This is what being President is really about.

In one sense, the July 4 pyrotechnics from the isolated state ushered in an alarming new reality, one Trump is the first President to face -- the prospect that in theory, Pyongyang could soon hit the US with a nuclear-capable missile.

But what makes Trump's job so difficult is the unpalatable set of options available to try to halt North Korea's nuclear and missile programs. If those fail, an equally unpleasant option would await -- accepting the reality the United States is in Pyongyang's crosshairs.

In other words, Trump is under intense pressure to solve what may be an insoluble foreign policy problem.

He would have to decide how to contain the threat from the North Korean program or to deter the use of a weapon, effectively accepting that in theory at last Pyongyang had the US in the crosshairs.

"There is an argument to be made that everything has changed and nothing has changed," said Jim Walsh, senior research associate at the Massachusetts Institute of Technology Security Studies Program, adding that the test had demonstrated new North Korean capabilities and taken the US across a psychological and political threshold.

But Walsh said the options for the US to respond "really haven't changed. Today's options are no different from yesterday's options, and yesterday's options weren't very good."

Given that military attempts to halt North Korea's nuclear march all risk a horrific confrontation that could kill millions on the Korean peninsula and beyond, Trump has few alternatives but to seek a diplomatic outcome to the showdown with Pyongyang.

Yet there are few approaches that other presidents have not already tried.

One route the administration is taking is a familiar one -- seeking a Security Council condemnation of the test launch on Wednesday at the UN and new sanctions on the already heavily sanctioned North. US and South Korean forces are also conducting exercises in the region in response to the test.

Trump came to office slamming the "strategic patience" strategy pursued by the previous Obama administration on North Korea -- involving tough sanctions and a refusal to talk to Pyongyang until it renounces nuclear development.

But he has yet to diverge substantially from the approach of the last few administrations.

His preferred initial tactic was also a familiar one -- a charm offensive to convince Chinese President Xi Jinping, whom he will meet in Germany, to pressure Beijing's recalcitrant neighbor into halting its nuclear and missile programs.

But now the President, only three months after meeting Xi at his Florida resort, appears to have concluded that effort has failed, further narrowing his options.

"North Korea has just launched another missile. Does this guy have anything better to do with his life?" Trump tweeted after Monday's launch.

"Hard to believe that South Korea and Japan will put up with this much longer. Perhaps China will put a heavy move on North Korea and end this nonsense once and for all!"

On Wednesday, Trump added: "Trade between China and North Korea grew almost 40% in the first quarter. So much for China working with us but we had to give it a try!"

There are differing interpretations as to how much Beijing has so far done to pressure the North Koreans, following its decision to halt coal exports to the Stalinist state and a temporary freeze on oil supplies.

And Washington may be overestimating China's capacity to change the behavior of the volatile North Korean leader.

Many experts also believe that China is reluctant to try the kind of prolonged oil embargo that could really pressure Kim because of a fear it could collapse his regime and ignite a chaotic situation on the peninsula. Beijing also has no interest in a solution that would lead to a unified Korea in alliance with the US on its borders.

Whatever China's motivations, however, it seems unlikely that its leaders will be swayed by Trump tweets, hence the need for a prolonged and comprehensive diplomatic push by the administration starting at the G20.

"This is really a good opportunity for the President to show leadership, and to show the type of leader he is," said Harry Kazianis, Director of Defense Studies at the Center for the National Interest, who argued that Trump's tweets were merely a type of "strategic signaling" not the extent of US policy.

If Trump's powers of persuasion with other world leaders fall short, he and his administration will be left with some tough decisions.

One option would be to expand secondary sanctions on Chinese firms that do business with North Korea to try to tighten an economic chokehold around Pyongyang.

"We really need to focus on the players in China, Chinese banks. the people that are aiding and abetting the money laundering," said Kazianis, who also advocates stepping up cybersecurity operations against Pyongyang's missile and nuclear programs.

Widening sanctions on China's firms is a logical next step, one of the few new approaches that the administration could pursue.

But such a move would also create a whole new foreign policy headache by triggering a sharp deterioration in relations with China, a scenario that could have unpredictable results and significantly increase regional tensions in Asia.

Another option for Trump -- actually talking to the North Koreans -- has been tried before, and is problematic, since Pyongyang has in the past agreed to nuclear freezes and walked away from the deal. Kim, having watched the demise of other dictators who gave away their nuclear programs, believes that his atomic weapons are the only guarantor of his survival.

"They will never give up their nuclear weapons program. They will never give up their missile program. That discussion is off the table," said Sue Mi Terry, a former CIA North Korea analyst, on CNN on Wednesday.

Whatever path he eventually chooses, it's clear that tough talk and tweets are unlikely to provide breakthroughs and that managing escalating tensions will consume the administration for as long as it is in office.

"The main danger here, contrary to some expectations, is not that North Korea is going to suddenly attack the US," Walsh said. "The danger is that there will be a war, but it will happen through miscalculation or misperception."

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Daily Stormer Troll Army Threatens CNN Staffers Over Reddit User Behind Trump/CNN GIF – Southern Poverty Law Center

Posted: at 10:45 pm

The events leading to this online call to arms began Sunday morning, whenPresident Trump tweeted a gif created by Reddit user HanAssholeSolo depicting a scene from Wrestlemania XXIII in which Trump body slams and pummels WWE promoter Vince McMahon. In the gif, the CNN logo is superimposed over McMahons face.

Auernheimer heralded the tweet as easily the greatest tweet in the history of Twitter.

After scouring HanAssholeSolo'sReddit account, which contained scores of racist and xenophobic postings, CNNs KFile was able to track down the users Facebook page and contact him.

Fearing public embarrassment and his safety, HanAssholeSolo published a lengthy apology on the Reddit group r/theDonald, asking that CNN not publish his identity. (The apology has since been removed.)

Alt-righter Jack Posobiec claims HanAssholeSolo is a 15 year-old LGBT person; however, CNN has identified the Reddit user as an adult male. (via Twitter)

CNN obliged, on the condition that HanAssholeSoloremove his offending posts and cease his trolling, but that didnt stop the self-proclaimed real media at the Daily Stormer from issuing an ultimatum to every staffer at CNN.

Just like CNN tracked down this child and used media exposure as a bludgeon against him for posting (truthful and funny) things that they dont like, we are going to begin tracking down their families as a bludgeon against them for publishing (seditiously fraudulent) things that we dont like, wrote Auernheimer. CNN, this is your one singular chance to walk back this behavior of public blackmail. You have one week to fix this.

Auernheimers list of demands includes the public firing of the KFile team, a denouncement of their alleged threats, a $50,000 college scholarship for HanAssholeSolo, and a public assurance that he and his family will never be harmed by your organization.

The only problem:HanAssholeSolo is an adult,according to CNN.

Andrew Kaczynski, senor editor of CNN's KFile, Tweets that HanAssholeSolo is an adult, not a teenager, as some have claimed. (via Twitter)

We are going to track down your parents. We are going to track down your siblings. We are going to track down your spouses. We are going to track down your children. Because hey, thats what you guys get to do, right? Were going to see how you like it when our reporters are hunting down your children, continued Auernheimer.

Auernheimer instructed CNN employees that do not want to be doxed to quit within the week and denounce the organizations alleged blackmail.

We didnt make these rules you did and now were going to force you to play by them. Hope you enjoy what is coming, you filthy rat kike bastards. Kill yourselves, kike news fakers. You deserve every single bit of what you are about to get, concluded Auernheimer.

The call to "kill the lying mass of shit that is CNN" posted to 4chan's politically incorrect forum, /pol/.

Within hours, personal information for multiple CNN staffers and their family members -- alongside images and gifs of individuals with CNN superimposed over their facesbeing shot in the head -- appeared in the comments of the posting.

The incident is a rare moment of unity for the far-right with members of r/theDonald, 4chan, the Daily Stormer, and the alt-lite banding together to attack CNN.

The 4chan message board /pol/, which isdedicated topolitically incorrect discussion,dubbed the campaign Operation:Autism Storm and posted a four part plan of attack that includes banding together with other far right sites, going after CNNs advertisers, discrediting everyone at CNN, and forming a legal strategy for HanAssholeSolo should he later be doxed.

At least nine separate hashtags trended across far-right accounts Tuesday evening including #cnnblackmail, #cnndoxing, and #fraudnewscnn as the controversy erupted.

To people who troll on the Internet for fun, wrote HanAssholeSolo. Consider your words and actions conveyed in your message and who it might upset or anger. Put yourself in their shoes before you post it. If you have a problem with trolling it is an addiction just like any other addiction someone can have to something and dont be embarrassed to ask for help.

The Daily Stormers veteran troll army hasnt heeded his advice.

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