Monthly Archives: April 2014

Full Body Strength and Conditioning: Team DNA – Video

Posted: April 28, 2014 at 10:44 pm


Full Body Strength and Conditioning: Team DNA
Calisthenics and weight training go hand in hand if you #39;re trying to get bigger retain muscle control. This is a full body routine for beginners and intermediates that can be done in a very...

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Full Body Strength and Conditioning: Team DNA - Video

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New Flu Shot Contains Insect DNA – Video

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New Flu Shot Contains Insect DNA
http://experimentalvaccines.org/2014/04/27/new-flu-shot-contains-insect-dna/

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New Flu Shot Contains Insect DNA - Video

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Bubu si dna Daniela – Video

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Bubu si dna Daniela

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Bubu si dna Daniela - Video

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DNA test: Remains from Yemen airstrike not al Qaeda bomb-maker's

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By Paul Cruickshank, Mohammed Jamjoom and Nic Robertson, CN

updated 10:51 AM EDT, Mon April 28, 2014

Strikes targeted al Qaeda fighters in Yemen.

STORY HIGHLIGHTS

(CNN) -- The remains of a Saudi national killed in airstrikes in Yemen earlier this month are not those of a wanted al Qaeda bomb-maker, according to multiple sources in Saudi Arabia who were briefed on the matter.

DNA tests conducted by Saudi officials showed that the remains were not those of Ibrahim al-Asiri, they said.

Saudi officials had obtained a close match to al-Asiri's DNA via remains of his brother, who died in a failed suicide bomb attack. The brother had carried a bomb inside his body, which killed him upon detonation but failed to hit his intended target, Saudi Arabia's security chief.

The sources said that the results were also negative for a DNA match to Nasser al-Wuhayshi, believed to be head of al Qaeda in the Arabian Pennisula. Al-Wuhayshi has been referred to as the "crown prince" of the global terror organization al Qaeda.

Crackdown

The laboratory tests were conducted to determine whether a broad offensive against AQAP, which is considered al Qaeda's most dangerous wing, had eliminated the two men.

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DNA test: Remains from Yemen airstrike not al Qaeda bomb-maker's

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DNA test: Remains not al Qaeda bomb-maker

Posted: at 10:44 pm

The remains of a Saudi national killed in airstrikes in Yemen earlier this month are not those of a wanted al Qaeda bomb-maker, according to multiple sources in Saudi Arabia who were briefed on the matter.

DNA tests conducted by Saudi officials showed that the remains were not those of Ibrahim al-Asiri, they said.

Saudi officials had obtained a close match to al-Asiri's DNA via remains of his brother, who died in a failed suicide bomb attack. The brother had carried a bomb inside his body, which killed him upon detonation but failed to hit his intended target, Saudi Arabia's security chief.

The sources said that the results were also negative for a DNA match to Nasser al-Wuhayshi, believed to be head of al Qaeda in the Arabian Pennisula. Al-Wuhayshi has been referred to as the "crown prince" of the global terror organization al Qaeda.

Crackdown

The laboratory tests were conducted to determine whether a broad offensive against AQAP, which is considered al Qaeda's most dangerous wing, had eliminated the two men.

The crackdown began over a week ago, resulting in the deaths of more than 60 suspected al Qaeda militants in Yemen from April 19 t0 21, Yemeni officials have said.

Operations have included attacks by Yemeni commandos and suspected U.S. drone strikes, a high-level Yemeni government official said.

U.S. special operations troops helped during that offensive, in part by flying Yemeni forces to a remote, mountainous spot in southern Yemen, though no Americans took part in combat, a U.S. official said.

Terror threats

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DNA links serial rapist to two rapes from 1993

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CLEVELAND - A serial rapist is facing up to 50 years in prison for a pair of sexual attacks that happened more than 20 years ago.

DNA fingerprints linked 55-year-old Kenneth Parker to the rapes of two Cleveland women from April and June of 1993.

In both cases Parker forced the victims into vacant dwellings using weapons or the threat of a weapon.

Last week Parker entered a plea of guilty to three counts of rape and four counts of kidnapping for the Cleveland rapes.

Parker was sentenced byCuyahoga County Common Pleas Court Judge Steven E. Gall to spend 16 to 50 years in prison for those rapes.

Parker is currently serving 18 years in prison for two rapes he committed in Columbus in 1996.

Judge Gall ordered the new sentence to begin in August 2015 when he would have been released from the 18-year sentence.

But for the DNA evidence, the victims in this case might never have gotten justice, and this serial rapist would have been free next year to resume attacking innocent women, said Assistant County Prosecutor Maxwell Martin, who prosecuted Parker for the State of Ohio. Now this community will stay remain from Mr. Parkers depravity.

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Naked Mole Rats and the Secret to Longevity

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Newswise SAN DIEGO (April 28, 2014) Zoo-goers may marvel at their bare skin and wrinkles, but scientists are more interested in the long lives of the pale, toothy and nearly hairless rodents known as naked mole rats. With lifespans of up to 31 years, naked mole rats live decades longer than would be expected based on their size. By comparison, mice live at most four years.

A new study links the naked mole rats remarkable lifespan to a molecular chaperone protein known as HSP25. HSP25 and other chaperone proteins act like a tiny quality-control team within an animals cells, quickly eliminating incorrectly manufactured or damaged proteins before they can cause a problem. Researchers say understanding changes in the actions of HSP25 during aging could shed light on age-related diseases like Alzheimers and Parkinsons.

Using a variety of rodents, we found that the amount of HSP25 present in their tissues positively correlated with the animals maximum lifespan, said Karl Rodriguez, Ph.D., a postdoctoral fellow at the University of Texas Health Science Center at San Antonio who conducted the experiments. If we can understand how HSP25 levels are regulated, what its function is and how it contributes to cell health, we might find ways to use this protein to combat devastating age-related diseases.

The researchers compared HSP25 levels in naked mole rats to levels of the protein found in rodents with different maximum lifespans, from mice (four years) to guinea pigs (12 years) to Damaraland mole rats (20 years) and others in between.

In animals with higher levels of HSP25, having more of these quality-control proteins means they are primed to react when there is a problem, so they can quickly transport the faulty protein to cellular garbage dumps and maintain the health of the cell, said Rodriguez.

Many neurodegenerative diseases, including Alzheimers, Parkinsons and prion diseases, are caused by defective proteins that are allowed to proliferate and accumulate into dangerous structures called aggregates. Finding ways to safely increase a persons level of HSP27 (the human corollary to HSP25) could potentially help to prevent or treat such diseases, said Rodriguez.

Native to the horn of Africa, naked mole rats live underground in colonies with complex social structures akin to those of ants or bees. In addition to their noted longevity, they are remarkably resistant to cancer.

Naked mole rats also appear to remain spry and healthy even in the final years of their long lives, so they can potentially offer clues not only about longevity but also the overall maintenance of health.

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North Island-wide facial eczema warning

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North Island farmers have been warned to check their stock for signs of facial eczema following a sharp jump in spore numbers from the fungus that causes this disease among livestock.

The disease is caused by spores from the fungus Pithomyces chartarum, which live in pasture and produce a spore containing a toxin that causes liver and bile-duct damage to livestock when eaten.

The high spore counts were the result of high soil temperatures and recent wet weather, AsureQuality facial eczema monitoring co-ordinator Leo Cooney said.

''There is a combination there that is a recipe for disaster.''

The most recent report from Gribbles Veterinary Laboratory on April 17 showed counts were at extremely high levels in many districts.

Most Waikato districts were rated as a high risk.

The district with the highest levels was the Hauraki Plains, which scored a count of 535,000 spores per gram followed by Waitomo, which scored 205,000 per gram.

The highest in the North Island was Horowhenua with 762,000 per gram.

Counts become dangerous to livestock when they measure over 100,000.

Cooney said the worst cases of facial eczema always followed droughts and these areas were the ones affected the most.

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Psoriasis On The Scalp – How To Treat Psoriasis! – Video

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Psoriasis On The Scalp - How To Treat Psoriasis!
If you suffer with psoriasis, you know it is the breakouts and the symptoms that can drive you crazy. The itching, the sores, the weeping skin; it is all a lot to bear. Psoriasis on the scalp...

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Scientists Hunt Down Origin of Huntington's Disease in the Brain and Provide Insights to Help Deliver Therapy

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Newswise The gene mutation that causes Huntingtons disease appears in every cell in the body, yet kills only two types of brain cells. Why? UCLA scientists used a unique approach to switch the gene off in individual brain regions and zero in on those that play a role in causing the disease in mice.

Published in the April 28 online edition of Nature Medicine, the research sheds light on where Huntingtons starts in the brain. It also suggests new targets and routes for therapeutic drugs to slow the devastating disease, which strikes an estimated 35,000 Americans.

From day one of conception, the mutant gene that causes Huntingtons appears everywhere in the body, including every cell in the brain, explained X. William Yang, professor of psychiatry and biobehavioral sciences at the Semel Institute for Neuroscience and Human Behavior at UCLA. Before we can develop effective strategies to treat the disorder, we need to first identify where it starts and how it ravages the brain.

Huntington's disease is passed from parent to child through a mutation in a gene called huntingtin. Scientists blame a genetic stutter -- a repetitive stretch of DNA at one end of the altered genefor the cell death and brain atrophy that progressively deprives patients of their ability to move, speak, eat and think clearly. No cure exists, and people with aggressive cases may die in as little as 10 years.

Huntingtons disease targets cells in two brain regions for destruction: the cortex and the striatum. Far more neurons die in the striatuma cerebral region named after its striped layers of gray and white matter. But its unclear whether cortical neurons play a role in the disease, including striatal neurons malfunction and death.

Yangs team used a unique approach to uncover where the mutant gene wreaks the most damage in the brain.

In 2008, Yang collaborated with co-first author Michelle Gray, a former UCLA postdoctoral researcher now at the University of Alabama, to engineer a mouse model for Huntingtons disease. The scientists inserted the entire human huntintin gene, including the stutter, into the mouse genome. As the animals brains atrophied, the mice developed motor and psychiatric-like problems similar to the human patients.

In the current study, Yang and Nan Wang, co-first author and UCLA postdoctoral researcher, took the model one step further. They integrated a genetic scissors that snipped off the stutter and shut down the defective genefirst in the cortical neurons, then the striatal neurons and finally in both sets of cells. In each case, they measured how the mutant gene influenced disease development in the cells and affected the animals brain atrophy, motor and psychiatric-like symptoms.

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